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development of IAA was considered probably as a result of oxidase deamination of NMT (Barker et al., 1980), but was afterwards recognized for being also partially by immediate deamination by MAO (Barker et al., 1982). Pretreatment Together with the MAO-I iproniazid in rat total Mind homogenates inhibited IAA formation by eighty three%, NMT and DMT-